| Non-homologous end joining as a mechanism of DNA repair
| [0163]
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| An antisense oligonucleotide targeted to human Ku80 messenger RNA sensitizes M059K malignant glioma cells to ionizing radiation, bleomycin, and etoposide but not DNA cross-linking agents
| [0163]
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| Mechanisms of enhancement of cytotoxicity in etoposide and ionising radiation-treated cells by the protein kinase inhibitor wortmannin
| [0163]
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| Oligonucleotide interactions. III.Conformational differences between deoxy-and ribodinucleoside phosphates
| [0163]
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| DNA looping by Ku and the DNA-dependent protein kinase
| [0163]
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| A means to a DNA end: The many roles of Ku
| [0163]
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| Vanillins--a novel family of DNA-PK inhibitors
| [0163]
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| A targeted chain-termination mutation in the mouse Ape gene results in multiple intestinal tumors
| [0163]
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| Sensing and repairing DNA double-strand breaks
| [0163]
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| Targeted expression of oncogenic K-ras in intestinal epithelium causes spontaneoustumorigenesis in mice
| [0163]
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| A targeted inhibition of DNA-dependent protein kinase sensitizes breast cancer cells following ionizing radiation
| [0163]
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| The tumor suppressor p53 can reduce stable transfection in the presence of irradiation
| [0163]
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| Modification of the ionizing radiation response in living cells byan scFv against the DNA-dependent protein kinase
| [0163]
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| Adenovirus-mediated heat-activated antisense Ku70 expression radiosensitizes tumor cells in vitro and in vivo
| [0163]
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| DNA repair and recombination factorRad51 is over-expressed in human pancreaticadenocarcinoma
| [0163]
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| Evidence of the involvement of p53 in gamma-radiation-induced DNA repair in human lymphoblasts.vint
| [0163]
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| Tranfert de gène pour modifier la réponse à la radiotherapie Cancer Radiother
| [0163]
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| A Ku80 fragment with dominant negative activity imparts a radiosensitive phenotype to CHO-K1 cells
| [0163]
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| Transfer of Ku80 RNA antisense decreases the radioresistance of human fibroblasts
| [0163]
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| DNA-dependent protein kinase catalytic subunit: structural requirements for kinase activity by DNA ends
| [0163]
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| Mechanism of interaction between Ku protein and DNA
| [0163]
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| DNA-PK and ATM are required for radiation-enhanced integration
| [0163]
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| Immunological disorders and DNA repair
| [0163]
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| In vitro and in vivo potentiation of radiosensitivity of malignant gliomas by antisense inhibition of the RAD51 gene
| [0163]
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| Ku proteins join DNA fragments as shown by atomic force microscopy
| [0163]
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| Silencing expression of the catalytic subunit of DNA-dependent protein kinase by small interfering RNA sensitizes human cells for radiation-induced chromosome damage, cell killing, and mutation
| [0163]
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| DNA double strand breaks (DSB) and non-homologous end joining (NHEJ) pathways in human leukemia
| [0163]
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| Genetic variants of NHEJ DNA ligase IV can affect the risk of developing multiple myeloma, a tumour characterised by aberrant class switch recombination
| [0163]
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| Selective inactivation of DNA-dependent protein kinase with antisense oligodeoxynucleotides: consequences for the rejoining of radiation-induced DNA double-strand breaks and radiosensitivity of human cancer cell lines
| [0163]
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| Ionizing radiation greatly improves gene transfer efficiency in mammalian cells
| [0163]
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| Radiation-induced recombination is dependent on KU80
| [0163]
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| DNA damaging agents improve stable gene transfer efficiency in mammalian cells
| [0163]
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| Modulation de la réponse cellulaire aux radiations ionisantes: vers de nouvelles cibles moléculaires
| [0163]
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| Structure of the Ku heterodimer bound to DNA and its implications for double-strand break repair
| [0163]
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| Characterization of the RNA binding properties of Ku protein
| [0163]
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| Photocross-linking of an oriented DNA repair complex. Ku bound at a single DNA end
| [0163]
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| Geometry of a complex formed by double strand break repair proteins at a single DNA end: recruitment of DNA-PKcs induces inward translocation of Ku protein
| [0163]
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| The solution structure of the DNA double-stranded break repair protein Ku and its complex with DNA: a neutron contrast variation study
| [0163]
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