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J. Med Chem.
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J. Med Chem.
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ACC
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ACS Med Chem Lett.
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Antiviral Res
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ACC
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AAC
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BMCL
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J. Virol.
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Nature Rev. Drug. Discov.
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The Art, Science and Technology ofPharmaceutical Compounding
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Dosage Calculations
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Remington: The Science and Practice of Pharmacy
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J. Biomater Sci. Polym. Ed.
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Pharm. Res.
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ACS Med Chem Lett
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IDRUGS
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J. Med. Chem.
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PINK1 protects against cell death induced by mitochondrial depolarization, by phosphorylating Bcl-xL and impairing its pro-apoptotic cleavage
| [0275]
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PTEN-inducible kinase 1 (PINK1)/Park6 is indispensable for normal heart function
| [0275]
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PINK1-phosphorylated mitofusin 2 is a Parkin receptor for culling damaged mitochondria
| [0275]
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Mitochondrial fusion is essential for organelle function and cardiac homeostasis
| [0275]
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Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin
| [0275]
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HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte
| [0275]
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The Parkinson's disease genes pink1 and parkin promote mitochondrial fission and/or inhibit fusion in Drosophila
| [0275]
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Small interfering RNA targeting the PINK1 induces apoptosis in dopaminergic cells SH-SY5Y
| [0275]
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The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations
| [0275]
|
Therapeutic potential and mechanism of kinetin as a treatment for the human splicing disease familial dysautonomia
| [0275]
|
PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65
| [0275]
|
Regulation of autophagy by metabolic and stress signaling pathways in the heart
| [0275]
|
Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
| [0275]
|
Mitotracker probes and mitochondrial membrane potential
| [0275]
|
Isolated mitochondria infusion mitigates ischemia-reperfusion injury of the liver in rats
| [0275]
|
Ablation of ALCAT1 mitigates hypertrophic cardiomyopathy through effects on oxidative stress and mitophagy
| [0275]
|
A molecular gate which controls unnatural ATP analogue recognition by the tyrosine kinase v-Src
| [0275]
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The mitochondrial intramembrane protease PARL cleaves human Pink1 to regulate Pink1 trafficking
| [0275]
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Parkin is recruited selectively to impaired mitochondria and promotes their autophagy
| [0275]
|
PINK1 is selectively stabilized on impaired mitochondria to activate Parkin
| [0275]
|
Wild-type PINK1 prevents basal and induced neuronal apoptosis, a protective effect abrogated by Parkinson disease-related mutations
| [0275]
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PINK1 Protects against Oxidative Stress by Phosphorylating Mitochondrial Chaperone TRAP1
| [0275]
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A zebrafish model of PINK1 deficiency reveals key pathway dysfunction including HIF signaling
| [0275]
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Engineering unnatural nucleotide specificity for Rous sarcoma virus tyrosine kinase to uniquely label its direct substrates
| [0275]
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Specific correction of a splice defect in brain by nutritional supplementation
| [0275]
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PARIS (ZNF746) repression of PGC-1alpha contributes to neurodegeneration in Parkinson's disease
| [0275]
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Mitochondrial PINK1--a novel cardioprotective kinase
| [0275]
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Loss of PINK1 Increases the Heart's Vulnerability to Ischemia-Reperfusion Injury
| [0275]
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A simple and fast experimental model of myocardial infarction in the mouse
| [0275]
|
PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility
| [0275]
|
Mechanisms of mitophagy
| [0275]
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Mitochondrial disease
| [0276]
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PINK1-Phosphorylated Mitofusin-2 Is a Parkin Receptor for Culling Damaged Mitochondria
| [0276]
|
PINK1 is selectively stabilized on impaired mitochondria to activate Parkin
| [0276]
|
PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility
| [0276]
|
Report of the 1995 World Health Organization/International Society and Federation of Cardiology Task Force on the Definition and Classification of cardiomyopathies
| [0276]
|
Harrison's Internal Medicine
| [0276]
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Wild-type PINK1 prevents basal and induced neuronal apoptosis, a protective effect abrogated by Parkinson disease-related mutations
| [0276]
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PINK1 as a molecular checkpoint in the maintenance of mitochondrial function and integrity
| [0276]
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Ubiquitin proteasome system in Parkinson's disease: a keeper or a witness
| [0276]
|
The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations
| [0276]
|
PARIS (ZNF746) repression of PGC-1alpha contributes to neurodegeneration in Parkinson's disease
| [0276]
|
Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis
| [0276]
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PINK1 Protects against Oxidative Stress by Phosphorylating Mitochondrial Chaperone TRAP1
| [0276]
|
Cytoplasmic Pink1 activity protects neurons from dopaminergic neurotoxin MPTP
| [0276]
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Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress
| [0276]
|
PINK1-linked parkinsonism is associated with Lewy body pathology
| [0276]
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Switching Cdk2 on or off with small molecules to reveal requirements in human cell proliferation
| [0276]
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Biochemical aspects of the neuroprotective mechanism of PTEN-induced kinase-1 (PINK1)
| [0276]
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Chemical Genetic Approach for Kinase-Substrate Mapping by Covalent Capture of Thiophosphopeptides and Analysis by Mass Spectrometry
| [0276]
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Covalent capture of kinase-specific phosphopeptides reveals Cdk1-cyclin B substrates
| [0276]
|
PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65
| [0276]
|
Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability
| [0276]
|
Cytokinin-induced differentiation of human myeloid leukemia HL-60 cells is associated with the formation of nucleotides, but not with incorporation into DNA or RNA
| [0276]
|
Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes
| [0276]
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DAP-kinase loss of expression in various carcinoma and B-cell lymphoma cell lines: possible implications for role as tumor suppressor gene
| [0276]
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LKB1 in lung cancerigenesis: a serine/threonine kinase as tumor suppressor
| [0276]
|
Recent advances in the genetics of Parkinson's disease
| [0276]
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Too many roads not taken
| [0276]
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Turning a protein kinase on or off from a single allosteric site via disulfide trapping
| [0276]
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Mechanism of action of A-769662, a valuable tool for activation of AMP-activated protein kinase
| [0276]
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Calcium-dependent protein kinase 1 is an essential regulator of exocytosis in Toxoplasma
| [0276]
|