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| PINK1 protects against cell death induced by mitochondrial depolarization, by phosphorylating Bcl-xL and impairing its pro-apoptotic cleavage
| [0275]
|
| PTEN-inducible kinase 1 (PINK1)/Park6 is indispensable for normal heart function
| [0275]
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| PINK1-phosphorylated mitofusin 2 is a Parkin receptor for culling damaged mitochondria
| [0275]
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| Mitochondrial fusion is essential for organelle function and cardiac homeostasis
| [0275]
|
| Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin
| [0275]
|
| HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte
| [0275]
|
| The Parkinson's disease genes pink1 and parkin promote mitochondrial fission and/or inhibit fusion in Drosophila
| [0275]
|
| Small interfering RNA targeting the PINK1 induces apoptosis in dopaminergic cells SH-SY5Y
| [0275]
|
| The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations
| [0275]
|
| Therapeutic potential and mechanism of kinetin as a treatment for the human splicing disease familial dysautonomia
| [0275]
|
| PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65
| [0275]
|
| Regulation of autophagy by metabolic and stress signaling pathways in the heart
| [0275]
|
| Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
| [0275]
|
| Mitotracker probes and mitochondrial membrane potential
| [0275]
|
| Isolated mitochondria infusion mitigates ischemia-reperfusion injury of the liver in rats
| [0275]
|
| Ablation of ALCAT1 mitigates hypertrophic cardiomyopathy through effects on oxidative stress and mitophagy
| [0275]
|
| A molecular gate which controls unnatural ATP analogue recognition by the tyrosine kinase v-Src
| [0275]
|
| The mitochondrial intramembrane protease PARL cleaves human Pink1 to regulate Pink1 trafficking
| [0275]
|
| Parkin is recruited selectively to impaired mitochondria and promotes their autophagy
| [0275]
|
| PINK1 is selectively stabilized on impaired mitochondria to activate Parkin
| [0275]
|
| Wild-type PINK1 prevents basal and induced neuronal apoptosis, a protective effect abrogated by Parkinson disease-related mutations
| [0275]
|
| PINK1 Protects against Oxidative Stress by Phosphorylating Mitochondrial Chaperone TRAP1
| [0275]
|
| A zebrafish model of PINK1 deficiency reveals key pathway dysfunction including HIF signaling
| [0275]
|
| Engineering unnatural nucleotide specificity for Rous sarcoma virus tyrosine kinase to uniquely label its direct substrates
| [0275]
|
| Specific correction of a splice defect in brain by nutritional supplementation
| [0275]
|
| PARIS (ZNF746) repression of PGC-1alpha contributes to neurodegeneration in Parkinson's disease
| [0275]
|
| Mitochondrial PINK1--a novel cardioprotective kinase
| [0275]
|
| Loss of PINK1 Increases the Heart's Vulnerability to Ischemia-Reperfusion Injury
| [0275]
|
| A simple and fast experimental model of myocardial infarction in the mouse
| [0275]
|
| PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility
| [0275]
|
| Mechanisms of mitophagy
| [0275]
|
| Mitochondrial disease
| [0276]
|
| PINK1-Phosphorylated Mitofusin-2 Is a Parkin Receptor for Culling Damaged Mitochondria
| [0276]
|
| PINK1 is selectively stabilized on impaired mitochondria to activate Parkin
| [0276]
|
| PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility
| [0276]
|
| Report of the 1995 World Health Organization/International Society and Federation of Cardiology Task Force on the Definition and Classification of cardiomyopathies
| [0276]
|
| Harrison's Internal Medicine
| [0276]
|
| Wild-type PINK1 prevents basal and induced neuronal apoptosis, a protective effect abrogated by Parkinson disease-related mutations
| [0276]
|
| PINK1 as a molecular checkpoint in the maintenance of mitochondrial function and integrity
| [0276]
|
| Ubiquitin proteasome system in Parkinson's disease: a keeper or a witness
| [0276]
|
| The PINK1/Parkin-mediated mitophagy is compromised by PD-associated mutations
| [0276]
|
| PARIS (ZNF746) repression of PGC-1alpha contributes to neurodegeneration in Parkinson's disease
| [0276]
|
| Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis
| [0276]
|
| PINK1 Protects against Oxidative Stress by Phosphorylating Mitochondrial Chaperone TRAP1
| [0276]
|
| Cytoplasmic Pink1 activity protects neurons from dopaminergic neurotoxin MPTP
| [0276]
|
| Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress
| [0276]
|
| PINK1-linked parkinsonism is associated with Lewy body pathology
| [0276]
|
| Switching Cdk2 on or off with small molecules to reveal requirements in human cell proliferation
| [0276]
|
| Biochemical aspects of the neuroprotective mechanism of PTEN-induced kinase-1 (PINK1)
| [0276]
|
| Chemical Genetic Approach for Kinase-Substrate Mapping by Covalent Capture of Thiophosphopeptides and Analysis by Mass Spectrometry
| [0276]
|
| Covalent capture of kinase-specific phosphopeptides reveals Cdk1-cyclin B substrates
| [0276]
|
| PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65
| [0276]
|
| Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability
| [0276]
|
| Cytokinin-induced differentiation of human myeloid leukemia HL-60 cells is associated with the formation of nucleotides, but not with incorporation into DNA or RNA
| [0276]
|
| Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes
| [0276]
|
| DAP-kinase loss of expression in various carcinoma and B-cell lymphoma cell lines: possible implications for role as tumor suppressor gene
| [0276]
|
| LKB1 in lung cancerigenesis: a serine/threonine kinase as tumor suppressor
| [0276]
|
| Recent advances in the genetics of Parkinson's disease
| [0276]
|
| Too many roads not taken
| [0276]
|
| Turning a protein kinase on or off from a single allosteric site via disulfide trapping
| [0276]
|
| Mechanism of action of A-769662, a valuable tool for activation of AMP-activated protein kinase
| [0276]
|
| Calcium-dependent protein kinase 1 is an essential regulator of exocytosis in Toxoplasma
| [0276]
|